目的 观察角膜创伤后，TCRδ-/-小鼠角膜神经末梢修复情况。推测γδ T细胞在角膜神经末梢再生中的作用。
方法 选取C57小鼠和γδT细胞缺陷小鼠TCRδ-/-各4只，刮除角膜上皮。96小时后，小鼠处死，取角膜进行免疫荧光染色。用anti-tubulin III-PE嗜染神经末梢，用DAPI嗜染角膜上皮细胞。角膜平铺制片后，去卷积显微镜下观察计数，对所有数据进行统计学分析，评价角膜神经末梢修复情况。
结果 γδT细胞缺陷组小鼠角膜神经末梢修复较差，神经末梢计数明显低于正常组C57小鼠( P < 0. 01)。
结论 角膜神经的修复过程中依赖γδT细胞的存在，该细胞可能是通过介导角膜早期炎症反应促进神经末梢的修复。
Objective The contribution of  TCRδ-/- mice to sensory Corneal nerve regeneration was investigated in C57 wildtype murine and TCRδ-/- mice. 
Methods using a model of corneal abrasion that removes the stratified epithelium and subbasalar nerve plexus,Point counting analysis of sub-basal nerve density in wound-related corneal regions at 96 hours after central epithelial abrasion comparing wildtype and TCRδ-/- mice, (n=4, means ± SEM).
Result In TCRδ-/- mice, there was a 30% attenuation compared with C57 wildtype mouse.
Conclusion These results document a beneficial role for the Corneal nerve regeneration depending on gamma delta T cell  related inflammatory response to corneal epithelial abrasion. Gamma Delta T cell  is necessary for Efficient   Corneal Nerve Regeneration.